In this case, root development (biologic eruption status), tooth position, and physical obstruction (radiographically evident or not) should be evaluated. Continue reading
DTE is often seen in the region of the maxillary canines. The maxillary canine develops high in the maxilla and is the only tooth that must descend more than its length to reach its position in the dental arch. When pathologic conditions are ruled out, the etiology of DTE of the canines has been suggested to be multifactorial. Specifically, 3 factors have been proposed for consideration: Continue reading
Other systemic conditions associated with impairment of growth, such as anemia (hypoxic hypoxia, histotoxic hypoxia, and anemic hypoxia) and renal failure, have also been correlated with DTE and other abnormalities in dentofacial development. Continue reading
Mucosal barrier has also been suggested as an etiologic factor in DTE. Any failure of the follicle of an erupting tooth to unite with the mucosa will entail a delay in the breakdown of the mucosa and constitute a barrier to emergence. Histologic studies have shown differences in the submucosa between normal tissues and tissues with a history of trauma or surgery. Gingival hyperplasia resulting from various causes (hormonal or hereditary causes, vitamin C deficiency, drugs such as phenytoin) might cause an abundance of dense connective tissue or acellular collagen that can be an impediment to tooth eruption. Continue reading
Primary or idiopathic failure of eruption is a condition described by Profitt and Vig, whereby nonankylosed teeth fail to erupt fully or partially because of malfunction of the eruption mechanism. This occurs even though there seems to be no barrier to eruption, and the phenomenon is considered to be due to a primary defect in the eruptive process. Terms such as arrested eruption and noneruption have been used interchangeably to describe a clinical condition that might have represented ankylosis, impaction, or idiopathic failure of eruption. These terms refer more to the pathogenesis of DTE than to the benchmarks that define DTE. Continue reading